Citation: Junbo Jia, Zhen Jin, Lili Chang, Xinchu Fu. Structural calculations and propagation modeling of growing networks based on continuous degree[J]. Mathematical Biosciences and Engineering, 2017, 14(5&6): 1215-1232. doi: 10.3934/mbe.2017062
[1] | Junbo Jia, Wei Shi, Pan Yang, Xinchu Fu . Immunization strategies in directed networks. Mathematical Biosciences and Engineering, 2020, 17(4): 3925-3952. doi: 10.3934/mbe.2020218 |
[2] | Meili Tang, Qian Pan, Yurong Qian, Yuan Tian, Najla Al-Nabhan, Xin Wang . Parallel label propagation algorithm based on weight and random walk. Mathematical Biosciences and Engineering, 2021, 18(2): 1609-1628. doi: 10.3934/mbe.2021083 |
[3] | Xiaonan Chen, Suxia Zhang . An SEIR model for information propagation with a hot search effect in complex networks. Mathematical Biosciences and Engineering, 2023, 20(1): 1251-1273. doi: 10.3934/mbe.2023057 |
[4] | A. N. Licciardi Jr., L. H. A. Monteiro . A complex network model for a society with socioeconomic classes. Mathematical Biosciences and Engineering, 2022, 19(7): 6731-6742. doi: 10.3934/mbe.2022317 |
[5] | Zhen Jin, Guiquan Sun, Huaiping Zhu . Epidemic models for complex networks with demographics. Mathematical Biosciences and Engineering, 2014, 11(6): 1295-1317. doi: 10.3934/mbe.2014.11.1295 |
[6] | Angel Martin-del Rey . A novel model for malware propagation on wireless sensor networks. Mathematical Biosciences and Engineering, 2024, 21(3): 3967-3998. doi: 10.3934/mbe.2024176 |
[7] | Jinna Lu, Xiaoguang Zhang . Bifurcation analysis of a pair-wise epidemic model on adaptive networks. Mathematical Biosciences and Engineering, 2019, 16(4): 2973-2989. doi: 10.3934/mbe.2019147 |
[8] | Haoyu Wang, Xihe Qiu, Jinghan Yang, Qiong Li, Xiaoyu Tan, Jingjing Huang . Neural-SEIR: A flexible data-driven framework for precise prediction of epidemic disease. Mathematical Biosciences and Engineering, 2023, 20(9): 16807-16823. doi: 10.3934/mbe.2023749 |
[9] | Anna Cattani . FitzHugh-Nagumo equations with generalized diffusive coupling. Mathematical Biosciences and Engineering, 2014, 11(2): 203-215. doi: 10.3934/mbe.2014.11.203 |
[10] | Meng Zhao, Wan-Tong Li, Yang Zhang . Dynamics of an epidemic model with advection and free boundaries. Mathematical Biosciences and Engineering, 2019, 16(5): 5991-6014. doi: 10.3934/mbe.2019300 |
Complex networks are good models for describing and studying complex systems [6,1,21]. It ignores many properties that are not related to the study, and describes the system as a graph containing only nodes and edges, in which nodes represent the elements of the system and edges represent the interactions between them. Some real world networks include the WWW(World Wide Web) networks [8], Internet networks, collaboration networks, citation networks, metabolism networks etc, and some constructed networks include Euler graph, 'small world' network [25], BA network [3], etc.
To study complex networks and their properties, we should first study some basic special quantities characterizing the topology structure of the networks. Traditionally, these characteristic quantities are borrowed from graph theory, such as degree distribution
Transmission dynamics on complex networks is another focus of network research. Its dynamical behavior is often influenced by the network topology. Regarding the spread of epidemic, for example, different network topologies often have different threshold and propagation behavior. The main existing epidemic models are node-based model [22,26,0], pairwise models [11,12], effective degree models [15], and edge-based models [20,19,18,24]. Although these models have different styles, they all divide the nodes and edges into different classes according to the node degree. Here we also consider the node degree as a continuous variable, and apply the CDM to establish a continuous degree SIS epidemic model on static BA network. Simultaneously, we also take into account the evolution of the network, and build a continuous degree SIS epidemic model on a BA growing network.
The rest of this paper is organized as follows. In Section 2, applying the CDM, we calculate the degree distribution of three different growing models, which are the BA growing model, the preferential attachment accelerating growing model and the random attachment growing model. In Section 3, we calculate the joint degree distribution of the BA model also using the CDM, and study its degree correlation. In Section 4, we establish a continuous degree SIS model on a static degree uncorrelated network and a BA growing network, respectively. Finally, in Section 5, we conclude the paper.
In this section, we use CDM to calculate the degree distribution of the following three growing models: the BA growing model, the preferential attachment accelerating growing model and the random attachment growing model. The main notations are defined in Table 1.
Notation | Definition |
| The total number of nodes in the initial network. |
| The total number of edges in the initial network. |
| The probability for the node |
| The total number of nodes at time |
| The total number of nodes which degree not more than |
| The cumulative distribution function of node degree, or the proportion of nodes which degree not more than |
| The degree distribution, or the probability density of node which degree equal to |
| The total number of directed edges at time |
| The total number of directed edges which degree sequentially not larger than |
| The joint cumulative distribution function at time |
| The joint degree distribution at time |
| The conditional degree distribution at time |
| The marginal distribution at time |
| The cumulative distribution function of susceptible nodes at time |
| The cumulative distribution function of infected nodes at time |
| The probability density of susceptible nodes which degree equal to |
| The probability density of infected nodes which degree equal to |
| The probability that a edge emitted by degree |
| The probability that a edge points to an infected node in degree unrelated network. |
First, we recall the BA model, whose evolution mechanism contains two aspects:
H1: Growth: Starting with a small number (
H2: Preferential attachment: the probability
We consider that the time
ˆN(k,t)=N(t)F(k,t)=N(t)∫k0p(x,t)dx. | (1) |
It's not difficult to find that the growth of the BA model satisfies the Markov property. That is to say, the network structure at time
For a node with degree
ˆN(k,t)+ΔˆN(k,t)=ˆN(k+θkt,t+Δt). | (2) |
(2) presents the quantitative relation between
N(t)F(k,t)+Δt=N(t+Δt)F(k+mΔtk2(l0+mt),t+Δt). | (3) |
According to the evolution mechanism of the model, we have
(m0+t)F(k,t)+Δt=(m0+t+Δt)F(k+mΔtk2(l0+mt),t+Δt). | (4) |
Taking the Taylor expansion for
mΔtk2(l0+mt)∂F(k,t)∂k+Δt∂F(k,t)∂t+Δt(F(k,t)−1)m0+t=o(Δt). | (5) |
Dividing by
mk2(l0+mt)∂F(k,t)∂k+∂F(k,t)∂t+F(k,t)−1m0+t=0, | (6) |
in the feasible region
{F(k,0)=Ψ(k),F(m,t)=0,∀t>0, | (7) |
where,
F(k,t)={1−1m0+t(m0+ml0+m2tk2−l0m), if m≤k≤m(1+mtl0)12;tm0+t+m0m0+tΨ(k(1+mtl0)12), if k>m(1+mtl0)12. | (8) |
This equation means that, for given time
Assuming that the derivative of cumulative distribution function
p(k,t)=∂F(k,t)∂k={2ml0+2m2tm0+tk−3, if m≤k≤m(1+mtl0)12;m0(m0+t)(1+mtl0)12p(k(1+mtl0)12,0), if k>m(1+mtl0)12. | (9) |
As we can see,
We assume
⟨kt⟩=∫∞mkp(k,t)dk=2(l0+mt)m0+t. |
From this expression, we can easily understand that the average degree
Different from BA model, many real networks exhibit accelerating growth, such as the Internet and WWW [8,23,5,9], where the edge number grows faster than the nodes numbers. Here we use the CDM to calculate the degree distribution of one preferential attachment accelerating growing model with
Let
N(t)F(k,t)+Δt=N(t+Δt)F(k+θkt,t+Δt), | (10) |
where
(α+1)k2t∂F(k,t)∂k+∂F(k,t)∂t+F(k,t)−1m0+t=0, | (11) |
in the feasible region
F(mtα,t)=0,∀t>0. | (12) |
Solving this boundary problem, we obtain
F(k,t)=1−1m0+t(m0+(km)2α−1t1+α1−α). | (13) |
Consequently, the degree distribution is
p(k,t)=∂F(k,t)∂k=2(m0+t)(1−α)m21−αt1+α1−αk−3−α1−α≈21−αm21−αt2α1−αk−3−α1−α. | (14) |
This result is completely equivalent to (24) in [23]. The simulation results and analytical results are showed in Figure 2.
In this subsection, we will calculate the degree distribution of random attachment growing model using CDM. Same as the BA model, the random attachment growing model also starts with a small number (
Similar to the BA growing model, we also have the following relationship on cumulative distribution function
N(t)F(k,t)+Δt=N(t+Δt)F(k+θkt,t+Δt), | (15) |
where
mm0+t∂F(k,t)∂k+∂F(k,t)∂t+F(k,t)−1m0+t=0, | (16) |
also in the feasible region
Solving (16), and substituting (7) into the general solution, we get
F(k,t)={1−e1−km,m<k≤m+ln(m0+tm0)m;tm0+t+m0m0+tΨ(k+ln(m0m0+t)m),k>m+ln(m0+tm0)m. | (17) |
If we ignore the initial
p(k,t)=∂F(k,t)∂k=1me1−km,m<k≤m+ln(m0+tm0)m. | (18) |
This result is the same as the (17) in [2], which verifies the accuracy of CDM again. In Figure 3, we show the simulation results as well as analytical results.
In Section 2, we have shown that the CDM can be used to calculate the degree distribution of some growing models. In this section, with the help of the CDM, we will calculate the joint degree distribution of BA model.
Assume that the edges have direction, so the total number of directed edges, written as
In the continuous degree case, let
ˆL(k1,k2,t)=L(t)F(k1,k2,t), | (19) |
F(k1,k2,t)=∫k10∫k20p(x1,x2,t)dx1dx2. | (20) |
For a directed edge which from one node with degree
ˆL(k1,k2,t)+ΔˆL(k1,k2,t)=ˆL(k1+θk1t,k2+θk2t,t+Δt). | (21) |
Substituting (19) into (21), we have
L(t)F(k1,k2,t)+ΔˆL(k1,k2,t)=L(t+Δt)F(k1+θk1t,k2+θk2t,t+Δt), | (22) |
where,
ΔL1=mΔt∫k1mkN(t)p(k,t)dk∫∞mkp(k,t)N(t)dk, | (23) |
and
ΔL2=mΔt∫k2mkN(t)p(k,t)dk∫∞mkp(k,t)N(t)dk. | (24) |
In the above two equations,
Substituting
2(l0+mt)F(k1,k2,t)+mΔt(2−mk1−mk2)=2(l0+mt+mΔt)F(k1+θk1t,k2+θk2t,t+Δt), | (25) |
where
mk12(l0+mt)∂F(k1,k2,t)∂k1+mk22(l0+mt)∂F(k1,k2,t)∂k2+∂F(k1,k2,t)∂t+ml0+mt(F(k1,k2,t)+m2k1+m2k2−1)=0, | (26) |
in the feasible region
{F(k1,m,t)=0,∀t>0,m<k1≤m(1+mtl0)12,F(m,k2,t)=0,∀t>0,m<k2≤m(1+mtl0)12. | (27) |
Using the characteristic line method to solve this boundary value problems, we get
F(k1,k2,t)=1−mk1−mk2+m2k1k2. | (28) |
This joint cumulative distribution function applies only to the directed edges between the young nodes. The proportion of these directed edges to the total directed edges is
Differentiating the (28) sequentially with respect to
p(k1,k2,t)=∂2F∂k2∂k1=m2k21k22. | (29) |
From (29) we can see that the joint degree distribution
For the joint degree distribution in (29), it is easy to verify that
1. Symmetry, namely
p(k1,k2)=p(k2,k1),∀k1,k2≥m, | (30) |
2. Normalization, namely
∫∞m∫∞mp(k1,k2)dk2dk1=1, | (31) |
3. Marginal distribution, namely
q(k2)=∫∞mp(k1,k2)dk1=mk22, | (32) |
where
Now, we consider the conditional degree distribution
p(k2|k1,t)=⟨kt⟩p(k1,k2,t)k1p(k1,t)=mk22. | (33) |
From (33), we know that in BA growing network the conditional degree distribution
Next, we study the degree correlation of BA model by using the assortativity coefficient
Ck1,k2=⟨k⟩N[k1k2]k1[k1]k2[k2]=1. | (34) |
From (34) we can get that the edge number of
In addition to the structure of the network, we will use the CDM to study the epidemic spreading on static and growing networks, respectively.
In order to establish a continuous degree SIS model on a static degree uncorrelated network, first, let each node exist only in two discrete states, susceptible or infected. We use
ˆN(k,t)=ˆNS(k,t)+ˆNI(k,t), | (35) |
ˆNS(k,t)=N(t)FS(k,t), | (36) |
ˆNI(k,t)=N(t)FI(k,t), | (37) |
FS(k,t)=∫k0pS(x,t)dx, | (38) |
FI(k,t)=∫k0pI(x,t)dx. | (39) |
Then we consider the spread mechanism. At each time step, one susceptible node becomes infected node due to the contact with the infected neighbors, and the probability of each contact and infection with infected nodes is
The epidemic spreading also satisfies the Markov property. So from time
{pS(k,t+Δt)=γΔtpI(k,t)+(1−λkΘkΔt)pS(k,t),pI(k,t+Δt)=(1−γΔt)pI(k,t)+λkΘkΔtpS(k,t), | (40) |
where,
{∂pS(k,t)∂t=−λkΘpS(k,t)+γpI(k,t),∂pI(k,t)∂t=λkΘpS(k,t)−γpI(k,t). | (41) |
This is the continuous degree SIS model on degree uncorrelated static networks. This model is corresponding to the discrete degree SIS model established by R. Pastor-Satorras and A. Vespignani in [22]. The essential difference is that the variable
Similar to the discrete degree SIS model, the spread threshold of epidemic is also equal to
It is very difficult to obtain the analytical solution of (41). Take the BA static network as an example, we perform the simulations. As depicted in Figure 5, the stochastic simulations are in good accord with the numerical simulations. In Figure 5, We can also see that the smaller the ratio
It is shown in Figure 6 that for a node the greater the degree, the greater the probability of being infected. This is easy to understand that the big degree nodes have more neighbors, so the infected neighbors are more, thus the ratio of infected will increase.
It is well known that many networks in reality are evolving constantly, not in the static state, such as the WWW and friends networks, which exist the addition or extinction of nodes and the disconnection or rewiring of edges. At the same time, the spread of epidemic is entangled with the network evolution. And the spread is also influenced by the network evolution. In this subsection we use the CDM to study the special BA growing network whose growth along simultaneously with the spread of epidemic, and establish the continuous degree SIS model on a BA growing network.
The evolution mechanism of growing network consists of three parts: the H1 H2 in Section 2.1 and the following H3. Note that the nodes before the time step
H3: Epidemic Spread: at time step
From the above evolution mechanism, we can know that before the time step
The notation
{ˆNS(k,t)−ΔtλˆLSk(t)+ΔtγˆNI(k,t)+Δt=ˆNS(k+θkt,t+Δt),ˆNI(k,t)+ΔtλˆLSk(t)−ΔtγˆNI(k,t)=ˆNI(k+θkt,t+Δt). | (42) |
Substituting (36), (37), and
{N(t)FS(k,t)−ΔtλN(t)∫kmpS(x,t)xΘdx+ΔtγN(t)FI(k,t)+Δt=N(t+Δt)FS(k+θkt,t+Δt),N(t)FI(k,t)+ΔtλN(t)∫kmpS(x,t)xΘdx−ΔtγN(t)FI(k,t)=N(t+Δt)FI(k+θkt,t+Δt). | (43) |
Taking the Taylor expansion for the right-hand side of this equations, both sides of equations divided by
{mk2(l0+mt)∂FS(k,t)∂k+∂FS(k,t)∂t+FS(k,t)−1m0+t+λ∫kmxps(x,t)Θdx−γFI(k,t)=0,mk2(l0+mt)∂FI(k,t)∂k+∂FI(k,t)∂t+FI(k,t)m0+t−λ∫kmxps(x,t)Θdx+γFI(k,t)=0. | (44) |
The sum of two equations in (44) is
mk2(l0+mt)∂(FS+FI)∂k+∂(FS+FI)∂t+(FS+FI)−1m0+t=0. | (45) |
Where
(44) contains the variables
{mk2(l0+mt)∂pS(k,t)∂k+∂pS(k,t)∂t=−λΘkpS(k,t)+γpI(k,t)−(m2(l0+mt)+1m0+t)pS(k,t),mk2(l0+mt)∂pI(k,t)∂k+∂pI(k,t)∂t=λΘkpS(k,t)−γpI(k,t)−(m2(l0+mt)+1m0+t)pI(k,t), | (46) |
where
{pI(k,t0)=p∗I(k),pS(k,t0)=p(k,t)−p∗I(k),pI(m,t)=0,∀t>t0,pS(m,t)=p(m,t),∀t>t0. | (47) |
As described in Section 4.1, the spread threshold of epidemic on static BA network is infinite, so the spread threshold of epidemic on BA growing network is also infinite if the growth is not very fast, namely, small amount of infected nodes can also cause the outbreak of epidemic.
Solving analytically these equations is difficult, so we perform the following simulation. In Figure 7, we find that the big degree nodes have a relatively high probability of being infected,
As depicted in Figure 8, the newly added health nodes can slightly reduce the ratio of infected nodes, but the final ratio of infected nodes will gradually tend to the final ratio of infected nodes of SIS model on static network. This result can also be obtained from (46), because that when time
When a network reaches certain size, the node degree can be considered as a continuous variable, so we put forward the CDM to calculate the degree distribution. Using the CDM we calculate the degree distribution of the three growing models, which are the BA growing model, the preferential attachment accelerating growing model with
Although the analytical solution of PDEs is usually difficult to obtain, there is some advantages of CDM, i.e., we can transform the evolution of network into a partial differential equation(s) on cumulative distribution function or probability density function, thus we can study analytically the structure of networks and the spread of epidemic on them.
In this paper, we use the CDM to calculate the topology structure of networks and establish the epidemic models on these networks. However, the evolution mechanism of networks in reality is very complicated. For example, a network may exist the addition or extinction of nodes, the disconnection or rewiring of edges, or even the both. When considering the state of nodes, the evolution of the nodes and edges may depend on the state of nodes, which are likely to occur in a real social network. E. g., the susceptible individuals may break the connection with infected, or rewire with other susceptible. Thus, with the CDM we will consider the following problems: (1) which topological structure can also be studied; (2) When we consider the state of nodes and the evolution mechanism of networks, how should we build the model in this case.
It is a pleasure to thank professor Zhenchao Han of Rutgers University for modification and guidance. This work was supported by National Natural Science Foundation of China under Grant Nos.11331009,11171314, TianYuan Special Foundation of the National Natural Science Foundation of China (11226259), Research Project supported by Shanxi Scholarship Council of China (2013-3), outstanding creative team of colleges and universities in Shanxi Province No. 232548901001, and sci-tech innovation team in Shanxi Province "The spread of infectious diseases prevention and control" No. 2015013001-06.
[1] | [ R. Albert,A. L. Barabási, Statistical mechanics of complex networks, Reviews of Modern Physics, 74 (2002): 47-97. |
[2] | [ A. L. Barabási,R. Albert,H. Jeong, Mean-field theory for scale-free random networks, Physica A: Statistical Mechanics and its Applications, 272 (1999): 173-187. |
[3] | [ A. L. Barabási,R. Albert, Emergence of scaling in random networks, Science, 286 (1999): 509-512. |
[4] | [ S. N. Dorogovtsev, J. F. F. Mendes and A. N. Samukhin, Structure of growing networks with preferential linking, Physical Review Letters, 85 (2000), 4633. |
[5] | [ S. N. Dorogovtsev and J. F. F. Mendes, Scaling properties of scale-free evolving networks: Continuous approach, Physical Review E, 63 (2001), 056125. |
[6] | [ S. N. Dorogovtsev and J. F. F. Mendes, Evolution of Networks: From Biological Nets to the Internet and WWW, Oxford University Press, New York, 2013. |
[7] | [ P. Erdős,A. Rényi, On the strength of connectedness of a random graph, Acta Mathematica Hungarica, 12 (1961): 261-267. |
[8] | [ M. Faloutsos,P. Faloutsos,C. Faloutsos, On power-law relationships of the internet topology, ACM SIGCOMM Computer Communication Review, 29 (1999): 251-262. |
[9] | [ M. J. Gagen and J. S. Mattick, Accelerating, hyperaccelerating, and decelerating networks, Physical Review E, 72 (2005), 016123. |
[10] | [ T. House,M. J. Keeling, Insights from unifying modern approximations to infections on networks, Journal of The Royal Society Interface, 8 (2011): 67-73. |
[11] | [ M. J. Keeling, The effects of local spatial structure on epidemiological invasions, Proceedings of the Royal Society of London. Series B: Biological Sciences, 266 (1999): 859-867. |
[12] | [ K. T. D. Ken,M. J. Keeling, Modeling dynamic and network heterogeneities in the spread of sexually transmitted diseases, Proceedings of the National Academy of Sciences, 99 (2002): 13330-13335. |
[13] | [ P. L. Krapivsky, S. Redner and F. Leyvraz, Connectivity of growing random networks, Physical Review Letters, 85 (2000), 4629. |
[14] | [ P. L. Krapivsky and S. Redner, Organization of growing random networks, Physical Review E, 63 (2001), 066123. |
[15] | [ J. Lindquist,J. Ma,P. van den Driessche,F. H. Willeboordse, Effective degree network disease models, Journal of Mathematical Biology, 62 (2011): 143-164. |
[16] | [ C. Liu, J. Xie, H. Chen, H. Zhang and M. Tang, Interplay between the local information based behavioral responses and the epidemic spreading in complex networks, Chaos: An Interdisciplinary Journal of Nonlinear Science, 25 (2015), 103111, 7 pp. |
[17] | [ S. Milgram, The small world problem, Psychology Today, 2 (1967): 60-67. |
[18] | [ J. C. Miller,A. C. Slim,E. M. Volz, Edge-based compartmental modelling for infectious disease spread, Journal of the Royal Society Interface, 9 (2012): 890-906. |
[19] | [ J. C. Miller,I. Z. Kiss, Epidemic spread in networks: Existing methods and current challenges, Mathematical Modelling of Natural Phenomena, 9 (2014): 4-42. |
[20] | [ Y. Moreno,R. Pastor-Satorras,A. Vespignani, Epidemic outbreaks in complex heterogeneous networks, The European Physical Journal B-Condensed Matter and Complex Systems, 26 (2002): 521-529. |
[21] | [ M. E. J. Newman, The structure and function of complex networks, SIAM Review, 45 (2003): 167-256. |
[22] | [ R. Pastor-Satorras and A. Vespignani, Epidemic spreading in scale-free networks, Physical Review Letters, 86 (2001), 3200. |
[23] | [ D. Shi, Q. Chen and L. Liu, Markov chain-based numerical method for degree distributions of growing networks, Physical Review E, 71 (2005), 036140. |
[24] | [ E. Volz, SIR dynamics in random networks with heterogeneous connectivity, Journal of Mathematical Biology, 56 (2008): 293-310. |
[25] | [ D. J. Watts,S. H. Strogatz, Collective dynamics of "small-world" networks, Nature, 393 (1998): 440-442. |
[26] | [ H. Zhang, J. Xie, M. Tang and Y. Lai, Suppression of epidemic spreading in complex networks by local information based behavioral responses, Chaos: An Interdisciplinary Journal of Nonlinear Science, 24 (2014), 043106, 7 pp. |
1. | Dongmei Fan, Guo-Ping Jiang, Yu-Rong Song, Yin-Wei Li, Guanrong Chen, Novel epidemic models on PSO-based networks, 2019, 477, 00225193, 36, 10.1016/j.jtbi.2019.06.006 | |
2. | Junbo Jia, Wei Shi, Pan Yang, Xinchu Fu, Immunization strategies in directed networks, 2020, 17, 1551-0018, 3925, 10.3934/mbe.2020218 |
Notation | Definition |
| The total number of nodes in the initial network. |
| The total number of edges in the initial network. |
| The probability for the node |
| The total number of nodes at time |
| The total number of nodes which degree not more than |
| The cumulative distribution function of node degree, or the proportion of nodes which degree not more than |
| The degree distribution, or the probability density of node which degree equal to |
| The total number of directed edges at time |
| The total number of directed edges which degree sequentially not larger than |
| The joint cumulative distribution function at time |
| The joint degree distribution at time |
| The conditional degree distribution at time |
| The marginal distribution at time |
| The cumulative distribution function of susceptible nodes at time |
| The cumulative distribution function of infected nodes at time |
| The probability density of susceptible nodes which degree equal to |
| The probability density of infected nodes which degree equal to |
| The probability that a edge emitted by degree |
| The probability that a edge points to an infected node in degree unrelated network. |
Notation | Definition |
| The total number of nodes in the initial network. |
| The total number of edges in the initial network. |
| The probability for the node |
| The total number of nodes at time |
| The total number of nodes which degree not more than |
| The cumulative distribution function of node degree, or the proportion of nodes which degree not more than |
| The degree distribution, or the probability density of node which degree equal to |
| The total number of directed edges at time |
| The total number of directed edges which degree sequentially not larger than |
| The joint cumulative distribution function at time |
| The joint degree distribution at time |
| The conditional degree distribution at time |
| The marginal distribution at time |
| The cumulative distribution function of susceptible nodes at time |
| The cumulative distribution function of infected nodes at time |
| The probability density of susceptible nodes which degree equal to |
| The probability density of infected nodes which degree equal to |
| The probability that a edge emitted by degree |
| The probability that a edge points to an infected node in degree unrelated network. |