Introduction to AIMS Special Issue “How do Gamma Frequency Oscillations and NMDA Receptors Contribute to Normal and Dysfunctional Cognitive Performance”

2014, Volume 1, Issue 2: 183-184. doi: 10.3934/Neuroscience.2014.2.183

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Editorial Special Issues

Cardiff University Brain Research Imaging Centre, School of Psychology, Cardiff University, Park Place, Cardiff 02920, UK.

Received: 08 August 2014 Accepted: 14 August 2014 Published: 19 September 2014

Citation: Suresh D Muthukumaraswamy. Introduction to AIMS Special Issue “How do Gamma Frequency Oscillations and NMDA Receptors Contribute to Normal and Dysfunctional Cognitive Performance”[J]. AIMS Neuroscience, 2014, 1(2): 183-184. doi: 10.3934/Neuroscience.2014.2.183

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[1]	Pinotsis D, Friston K. (2014) Gamma oscillations and neural field DCMs can reveal cortical excitability and microstructure. AIMS Neurosci 1: 18-38.
[2]	Cadonic C, Albensi BC. (2014) The role of dynamic columns in explaining Gamma-band synchronization and NMDA receptors in cognitive functions. AIMS Neurosci 1: 52-64.
[3]	Moss RA, Moss J. (2014) The role of dynamic columns in explaining gamma-band synchronization and NMDA receptors in cognitive functions. AIMS Neurosci 1: 65-88.
[4]	Moss RA, Moss J. (2014) Commentary on the Pinotsis and Friston neural fields DCM and the Cadonic and Albensi oscillations and NMDA receptors articles. AIMS Neurosci 2: 145-149.
[5]	Chambers AM, Payne JD. (2014) Neural plasticity and learning: The consequences of sleep. AIMS Neurosci 2: 150-155.
[6]	Pinault D. (2014) N-methyl D-aspartate receptor antagonists amplify network baseline gamma frequency (30-80 Hz) oscillations: Noise and signal. AIMS Neurosci 2: 156-169.

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© 2014 the Author(s), licensee AIMS Press. This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0)

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